The Silent Killer – The Causes (Part 1)

The Silent Killer – The Causes (Part 1)

The silent killer is upon us, a modern epidemic so potent that it is believed to be responsible for almost half of deaths occurring each year in the USA. In the last article we discussed what the silent killer is, the misconceptions surrounding it and posed some ideas as to possible solutions to stop it. (NB, if you missed the last article you can read it here, http://viphealthsecrets.com)

In this article we will discuss the possible causes of arteriosclerosis, the silent killer, and the reasons why it is imperative you thoroughly understand them.
But before taking up our discussion of ways to forestall the consequences of arteriosclerosis such as a heart attack, it might therefore be well to understand more clearly the basic physi­ology involved.

The Physiology Behind The Silent Killer

Let us start with a closer look at the arteries, the vessels that carry fresh blood from the heart to the billions of cells in our bodies that are in constant need of nourishment. Upon careful examination, we find that the arteries are not the simple tubes we have pictured them to be. Viewing them in cross section, we see that their structure is more like that of a garden hose, con­taining three layers of tissue in the walls.

In both the outer and the intermediate layers, there are tiny intrinsic blood vessels which nourish the artery itself. The thickness and exact composition of the three layers vary, depend­ing upon an artery’s size and location.

The Two Types of Arteriosclerosis

Of the changes that may occur in the arteries as a result of disease, there are two types which concern us here. Both kinds have traditionally been known by the general term, “arterio­sclerosis,” which means hardening or thickening of the arteries.

Actually, however, there are two kinds of hardening of the arteries. One occurs when calcium deposits in the middle layer of the artery cause it to become brittle and hard. For this rea­son, it is sometimes called a “pipe-stem” artery. Such calcifica­tion does not necessarily obstruct the blood flow, and is usually harmless from a clinical point of view.

The other type of change, on the other hand—and it is the more frequent one—has serious consequences. It consists of a thickening of the inner wall of the artery by deposits of fats: cholesterol (a fatty alcohol), fatty acids, and the like, together with calcium.

As these deposits grow, the passageways or canals of the arteries become narrower, much in the same way as the drain from your kitchen sink becomes clogged with grease deposits. The result is that less and less blood can flow through the nar­rowed opening to the tissues or organs that depend on it for life. Your “pipes” have become clogged.

The Consequences of Arteriosclerosis…

At the same time, the swelling of the lining cells and rough­ening of the inner surface provide sites for formation of blood clots inside the narrowed artery. If the blockage is complete in vital arteries that feed the heart muscle, a heart attack - or as physicians call it, a coronary thrombosis - occurs.

If this dis­aster occurs in the cerebral arteries of the brain, a “stroke,” sometimes called a heart attack in the head, results. When the small arteries of the kidneys are affected, Bright’s disease, formerly called “dropsy,” and other diseases ensue. But whether the thickening and blocking process takes place in the heart, head, or kidneys, it is essentially the same disease. Doctors refer to it as atherosclerosis.

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